MIRACULOUS RECOVERY OF ITP PATIENT WITHOUT SPLEEN REMOVAL
My wife was hospitalized with dangerously low platelet count of 1K and diagnosed to be suffering from ITP. The treatment started with very high dose of prednisone, followed by IVIg followed by an even heavier dose of prednisone followed by a chemo drug retuxon with no improvement. She also went through painful bone marrow biopsy. She was then scheduled for a high risk surgery to remove her spleen with no guarantee that it will cure the problem. That was a very desperate situation. And we were all praying to Almighty for a miracle to cure my wife.
In the meantime I had started my own research on the web to see what else can be done. During my search I happen to find cidpusa website and sent an email to Dr Imran stating the entire situation.
In the very first email Dr Imran diagnosed her to be suffering from H.Pylori and unless that bacteria is eradicated through antibiotics followed by IVIg treatment, nothing will work. This diagnosis, more or less, was rejected by our doctor but he went along with us because nothing was working anyway. After ten days of antibiotics she was given IVIg, again on my asking. Her platelet count started the move upward after the second dose of IVIg. For the first time in four weeks when we were told that her platelet count is 18K, my wife wept and we both prostrated to our Creator in humility. Next day it was 36K and she was released from the hospital just two days before her scheduled surgery…….It was indeed a miracle.
I must say that without the online advice, correct diagnose, diligent and frequent email support of Dr Imran, throughout this episode, my wife’s spleen would have been removed and she most likely would have been in worst shape than before. And, by the way, we had access to the best hospitals and doctors available in northern California.
Today, she is nearly off the prednisone and her platelet count is well within the normal range. We profusely thank Allah swt who by his mercy showed us the path.
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Mercury
sources and
toxicity ROCHESTER, NEW YORK. Mercury is a highly toxic metal associated with damage to the kidneys and central nervous system. Mercury vapour is emitted from volcanoes, coal-burning power stations, and municipal incinerators and returns to the earth through rain contaminated with metallic mercury. Metallic mercury is methylated to methyl mercury in oceans and lakes and enters the food chain via fish and other seafood. Long-lived predator fish such as shark, swordfish, tilefish, king mackerel, and pike and bass in fresh water are the main sources of methyl mercury. Dental amalgams are an important source of mercury vapour and the vaccine preservative thimerosal is a significant source of ethyl mercury. Researchers at the University of Rochester School of Medicine recently published a review of what is currently known about mercury toxicity. Among the highlights:
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ADA fighting the
mercury battle
GAITHERSBURG, MARYLAND.
The American Dental
Association (ADA) has
launched an advertising
campaign to discourage
patients from having
their amalgam (silver)
fillings removed. Many
patients and sometimes
even their physicians
believe that mercury,
the main component of
amalgams, plays a role
in promoting such varied
diseases as Alzheimer's,
multiple sclerosis, and
autism. The ADA says the
evidence is not there
and their Code of Ethics
forbids dentists from
advising their patients
that there could be a
link. Scientists at the
University of Milan
disagree with the ADA
and point out that
several studies have
confirmed that mercury
from amalgam dental
fillings does enter
tissues and that the
mercury content of
brain, thyroid, kidney,
and pituitary gland
tissue is proportional
to the number of amalgam
fillings. They conclude
that the health effects
of amalgam fillings are
not at all clear and
need further
investigation. German
researchers point out
that some of the
composite materials used
in the replacement of
amalgam fillings may in
themselves be toxic.
Larkin, M. Don't
remove amalgam fillings,
urges American Dental
Association. The Lancet,
Vol. 360, August 3,
2002, p. 393